Unlikely. But seeing cosmic rays is common in space, very uncommon on earth.....
The cosmic ray I experienced one night while a high elevation (LASL - a summer job as a graduate student) was not the primary particle, probably not even one of the first or second generation daughters but they are all called cosmic rays too as in "cosmic ray shower." I have only seen it once while laying awake in bed, with eyes shut in a dark room, thinking about something else.
I am very interested in visual effects. I have twice see a very bright very small point of light that dimmed in a few seconds. I did have the presence of mind to with eyes still closed, to direct my direction of gaze (if my eyes were open) to different directions and noted the dying spot moved around. - I. e. I think a single retinal nerve in one eye, had some sort of "activity spasm." I spoke with a doctor or two at JHU's Wilmer Eye Institute as then I had many interactions with JHU doctors. They did not know what it was but agreed my understanding of it was probably correct. Also interesting was the perception was of a dimming bright spot on the ceiling of my bed room even with eyes still shut! All vision, is IMHO, a creation of the brain, not the image on the retina seen. The retina is just the major, but not the only, data base used for "seeing." Dreams with eyes shut prove this as do many experiments.
I am also quite familiar with phosphenes or "light flashes", but never have experienced any. I had modest contact with an NIH doctor, (Dr. Hambrick, if I recall his name correctly) who was doing some experiments with congenitally blind who wanted to help him (and perhaps themselves). I. e. small electrodes were placed on their visual cortex (V1) and when stimulated they reported phosphenes or "light flashes." Unfortunately these flashes continued for many seconds even after the electrical stimulation was turned off. Also if two near by electrodes were excited the spots "bloomed" into a region of phosphenes or "illuminated region" that could last for even a minute after the stimulation was turned off.
As it so happened, I was at the time stimulating the cerebellar area of a few Rhesus monkeys, which I am now sad to admit I and a neuro-surgeon of JHU had made have epilepsy with some brain damage in a prior operation. (A Doctor Copper in Boston was stimulating the cerebellar brain of humans and claiming he could prevent epilepsy and some other brain disorders – with every little animal experimental support for his theories) Thus, I knew there was a form of epilepsy, called Jacksonian epilepsy that starts out as very local excitation but spreads and then eventually dies out – I believe due to metabolic exhaustion of the active cells. It too can last for many seconds or even more than a minute. I suggested to Dr. Hambrick that he was triggering Jacksonian epilepsy and it would be impossible to give the blind even low resolution vision with his approach. Interestingly it is possible to give the blind low resolution vision by an array of many electrodes placed on their back or stomach. For proof read about Bach Rita's work of 20 years ago. (I exchanged a few Emails with him, but never met him in person. If that spelling is not correct, I will try to find his name and edit to spell it correctly for you.)
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The standard cognitive science “theory” of perception, including visual perceptions for which the most experimental data is available, is that perception “emerges” after many stages of neural transformations (Neural processing or calculations based on sensory input data) That is only “hand waving” - not enough of a neural mechanistic theory to even call it nonsense!
After many years of reading the literature on visual experiments, and a year of sabbatical leave from JHUAPL spent in the Cognitive Science department of JHU, nearly a dozen “hands on” experiments with humans and monkeys, I developed a theory of perception based on known properties of neurons which I call the RTS theory of perception.
(I had the great advantage of never having been taught how vision and perception are achieved.)
For vision, the first thing to explain (after simple sharp image formation on the retina and the electro-chemical processes convert photons into pulse in the optical nerve, is how is the continuous light field stimulating the retina neurons is parsed into discrete objects with boundaries in the field of continuous illumination. I suggested an explanation for that based on then known way near by “line detector neurons” in V1 mutually stimulate or inhibit each other. Then went on to suggest how these discrete objects are identified, etc. Explaining several of the Gestalt laws in neurological terms, not hand waving. I published all this in an obscure journal of APL (
The APL Technical Digest) very few in the field of cognitive science would read in 1991. I was requested to contribute an article on my RTS for an issue focused on various aspects of different simulations.
My Real Time Simulation theory is consistent with dozens of observation that contradict the cognitive science accepted “emergent” from input data POV and explains many known but seldom mentioned neurological facts, such as that more neural signals come to V1 (the “visual cortex”) from the parietal lobes than from the eyes, via the LNG. The RTS not only explains that, it RQUIRES that to be the case. The accepted theory of visual experience is shown to be false every time you have a visual dream. (Eyes shut – no input to transform into the visual experience.)
For more on the RTS please read:
http://www.sciforums.com/showthread.php?2868-About-determinism&p=882356&viewfull=1#post882356 but be warned it is long but most who do read think their time was well spent. It both explains and defends the RTS, especially how that POV makes genuine free will no longer conflict with the known DETERMINISTIC laws of what makes our nerves discharge.